Rapid effect of change in cerebrospinal fluid sodium concentration on salt appetite

@article{Weisinger1979RapidEO,
  title={Rapid effect of change in cerebrospinal fluid sodium concentration on salt appetite},
  author={Richard S. Weisinger and Phil Considine and Derek A. Denton and M J Mckinley and D R Mouw},
  journal={Nature},
  year={1979},
  volume={280},
  pages={490-491}
}
THIRST can be evoked within 1–2 min by intracarotid infusion of hypertonic NaCl. In contrast, the onset of a specific appetite for sodium salts as a result of rapid loss of body sodium is slow. A parotid fistula in naive sheep can produce a significant Na deficit (200–700 mmol) within 1–3 d but a Na appetite usually develops after 2–5 d (ref. 1). With large urinary water and Na loss induced by frusemide, water intake occurs within an hour but Na appetite is delayed until 24 h (ref. 2). Moreover… CONTINUE READING
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