Quantitative Trait Loci for Resistance to the Congenital Nephropathy in Tensin 2-Deficient Mice

@inproceedings{Sasaki2014QuantitativeTL,
  title={Quantitative Trait Loci for Resistance to the Congenital Nephropathy in Tensin 2-Deficient Mice},
  author={Hayato Sasaki and Nobuya Sasaki and Tomohiro Nishino and Ken-ichi Nagasaki and Hiroshi Kitamura and Daisuke Torigoe and Takashi Agui},
  booktitle={PloS one},
  year={2014}
}
The ICR-derived glomerulonephritis (ICGN) mouse is a chronic kidney disease (CKD) model that is characterized histologically by glomerulosclerosis, vascular sclerosis and tubulointerstitial fibrosis, and clinically by proteinuria and anemia, which are common symptoms and pathological changes associated with a variety of kidney diseases. Previously, we performed a quantitative trait locus (QTL) analysis to identify the causative genes for proteinuria in ICGN mice, and found a deletion mutation… CONTINUE READING

References

Publications referenced by this paper.
SHOWING 1-10 OF 41 REFERENCES

Similar Papers

Loading similar papers…