Protective effects of transduced PEP-1-Frataxin protein on oxidative stress-induced neuronal cell death.

Abstract

Reactive oxygen species (ROS) actively contribute to the development of a number of human diseases including ischemia. In response to oxidative stress, frataxin has a significant ability to improve cell survival though its biological function is unclear in relation to ischemia. To explore frataxin's role in protecting against ischemic cell death, we… (More)
DOI: 10.1016/j.jns.2010.08.016

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@article{Kim2010ProtectiveEO, title={Protective effects of transduced PEP-1-Frataxin protein on oxidative stress-induced neuronal cell death.}, author={Mi Jin Kim and Dae Won Kim and Ki-yeon Yoo and Eun Jeong Sohn and Hoon Jae Jeong and Hye Won Kang and Min Jea Shin and Eun Hee Ahn and Jae Jin An and Soon Won Kwon and Young Nam Kim and Moo-Ho Won and Sung-woo Cho and Jae-bong Park and Won Sik Eum and Soo Young Choi}, journal={Journal of the neurological sciences}, year={2010}, volume={298 1-2}, pages={64-9} }