Proinflammatory stimuli activates human-derived enteroglial cells and induces autocrine nitric oxide production.

@article{Cirillo2011ProinflammatorySA,
  title={Proinflammatory stimuli activates human-derived enteroglial cells and induces autocrine nitric oxide production.},
  author={Carla Cirillo and Giovanni Sarnelli and Fabio Turco and Annamaria Mango and Michela Grosso and Giovanni Aprea and Stefania Masone and Rosario Cuomo},
  journal={Neurogastroenterology and motility : the official journal of the European Gastrointestinal Motility Society},
  year={2011},
  volume={23 9},
  pages={
          e372-82
        }
}
BACKGROUND Enteric glial cells (EGCs) have been recently indicated as key regulators of intestinal inflammation in animals. Whether or not this is true and how these cells participate to inflammatory responses in humans is unknown. METHODS We isolated primary EGCs from human small bowel and then, we purified and characterized those using specific glial markers, such as S100B and glial fibrillary acidic protein (GFAP). To mimic an inflammatory scenario, we exposed EGCs to exogenous stimuli… CONTINUE READING
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