Progesterone potentiates IP(3)-mediated calcium signaling through Akt/PKB.


The activity of cells critically depends on the control of their cytosolic free calcium ion (Ca(2+)) concentration. The objective of the present study was to identify mechanisms of action underlying the control of the gain of intracellular Ca(2+) release by circulating gonadal steroid hormones. Acute stimulation of isolated neurons with progesterone led to… (More)
DOI: 10.1159/000113758


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