Production of interleukin (IL)-33 in the lungs during multiple antigen challenge-induced airway inflammation in mice, and its modulation by a glucocorticoid.

@article{Nabe2015ProductionOI,
  title={Production of interleukin (IL)-33 in the lungs during multiple antigen challenge-induced airway inflammation in mice, and its modulation by a glucocorticoid.},
  author={Takeshi Nabe and Hiroki Wakamori and Chihiro Yano and Ayumi Nishiguchi and Rino Yuasa and Hitomi Kido and Yusaku Tomiyama and Ayumi Tomoda and Haruka Kida and Anna Takiguchi and Masaya Matsuda and Keiichi Ishihara and Satoshi Akiba and Susumu Ohya and Hiroyuki Fukui and Nobuaki Mizutani and Shin Yoshino},
  journal={European journal of pharmacology},
  year={2015},
  volume={757},
  pages={34-41}
}
Although interleukin (IL)-33 is a candidate aggravator of asthma, the cellular sources of IL-33 in the lungs during the progression of antigen-induced airway inflammation remain unclear. Furthermore, it has not been determined whether the antigen-induced production of IL-33 can be pharmacologically modulated in vivo. In this study, we examined the… CONTINUE READING