Prevalence of neurofascin-155 antibodies in patients with multiple sclerosis

@article{Stich2016PrevalenceON,
  title={Prevalence of neurofascin-155 antibodies in patients with multiple sclerosis},
  author={Oliver Stich and S A Perera and Benjamin Berger and Sven Jarius and Brigitte Wildemann and A. Baumgartner and Sebastian Rauer},
  journal={Journal of the Neurological Sciences},
  year={2016},
  volume={364},
  pages={29-32}
}
From PNS to CNS: characteristics of anti-neurofascin 186 neuropathy in 16 cases
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Anti-neurofascin autoantibody and demyelination
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Preliminary evidence suggests that GBS/CIDP patients without detectable IgG antibodies on routine diagnostic tests may nevertheless benefit from IA, and that an unbiased screening approach using myelinating co-cultures may assist in the detection of further autoantibodies which remain to be identified in such patients.
Nodes of Ranvier during development and repair in the CNS
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The organization and function of CNS nodes of Ranvier are described and how these nodes change in demyelination and remyelinating disorders is considered, highlighting the impact of these changes on neuronal physiology in health and disease as well as the associated therapeutic implications.
Focal loss of the paranodal domain protein Neurofascin155 in the internal capsule impairs cortically induced muscle activity in vivo
Paranodal axoglial junctions are essential for rapid nerve conduction and the organization of axonal domains in myelinated axons. Neurofascin155 (Nfasc155) is a glial cell adhesion molecule that is

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