Prenatal particulate matter exposure and mitochondrial dysfunction at the maternal-fetal interface: Effect modification by maternal lifetime trauma and child sex.

@article{Brunst2018PrenatalPM,
  title={Prenatal particulate matter exposure and mitochondrial dysfunction at the maternal-fetal interface: Effect modification by maternal lifetime trauma and child sex.},
  author={Kelly J Brunst and Marco S{\'a}nchez-Guerra and Yueh-Hsiu Mathilda Chiu and Ander Wilson and Brent A. Coull and Itai Kloog and Joel Schwartz and Kasey J.M. Brennan and Michelle Bosquet Enlow and Robert O. Wright and Andrea A. Baccarelli and Rosalind J Wright},
  journal={Environment international},
  year={2018},
  volume={112},
  pages={
          49-58
        }
}
BACKGROUND Prenatal ambient fine particulate matter (PM2.5) and maternal chronic psychosocial stress have independently been linked to changes in mithochondrial DNA copy number (mtDNAcn), a marker of mitochondrial response and dysfunction. Further, overlapping research shows sex-specific effects of PM2.5 and stress on developmental outcomes. Interactions among PM2.5, maternal stress, and child sex have not been examined in this context. METHODS We examined associations among exposure to… CONTINUE READING

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Neuroendocrine Regulation of Air Pollution Health Effects: Emerging Insights.

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Prenatal Household Air Pollution Alters Cord Blood Mononuclear Cell Mitochondrial DNA Copy Number: Sex-Specific Associations

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