Prenatal exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin produces alterations in cortical neuron development and a long-term dysfunction of glutamate transmission in rat cerebral cortex.

@article{Tomasini2012PrenatalET,
  title={Prenatal exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin produces alterations in cortical neuron development and a long-term dysfunction of glutamate transmission in rat cerebral cortex.},
  author={Maria Cristina Tomasini and Sarah Beggiato and Luca Ferraro and Sergio Tanganelli and Luca Marani and Luca Lorenzini and Tiziana Antonelli},
  journal={Neurochemistry international},
  year={2012},
  volume={61 5},
  pages={759-66}
}
2,3,7,8-Tetrachlorodibenzo-p-dioxin (TCDD) is considered one of the most toxic dioxin-like compounds. It is ubiquitous in foodstuffs of animal origin and accumulates in the fatty tissues of animals and humans. Prenatal TCDD exposure has been associated, beside other effects, with persistent impaired cognitive development. In the present study, the effects of maternal exposure to TCDD during pregnancy on cortical neuron development at birth and cortical glutamate transmission in new-born, 14… CONTINUE READING

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