Prenatal exposure to bisphenols and cognitive function in children at 7 years of age in the Swedish SELMA study.

  title={Prenatal exposure to bisphenols and cognitive function in children at 7 years of age in the Swedish SELMA study.},
  author={Carl-Gustaf Bornehag and Elin Engdahl and Maria Unenge Hallerb{\"a}ck and Sverre Wikstr{\"o}m and Christian H. Lindh and Jo{\"e}lle R{\"u}egg and Eva Tanner and Chris Gennings},
  journal={Environment international},
Converging Effects of Three Different Endocrine Disrupters on Sox and Pou Gene Expression in Developing Rat Hippocampus: Possible Role of microRNA in Sex Differences
Endocrine disrupting chemicals (EDCs) can impair hippocampus-dependent behaviors in rat offspring and in children. In search for key processes underlying this effect, we compared the transcriptomes
Bisphenol A Inhibits the Transporter Function of the Blood-Brain Barrier by Directly Interacting with the ABC Transporter Breast Cancer Resistance Protein (BCRP)
It is demonstrated that BPA inhibits BCRP function in vitro, probably by direct interaction with the transporter, which might contribute to BPA’s known impact on neurodevelopment.
Association of bisphenol A, bisphenol F, and bisphenol S with ADHD symptoms in children.
  • Johanna Inhyang Kim, Young Ah Lee, Choong Ho Shin, Yun-Chul Hong, Bung-Nyun Kim, Youn-Hee Lim
  • Medicine
    Environment international
  • 2022


Prenatal exposure to bisphenol A and its alternatives and child neurodevelopment at 2 years.
Evidence is provided that prenatal exposure to BPA and BPS may affect child neuro development and that prenatal BPF exposure was not significantly associated with child neurodevelopment.
Exposure to bisphenol A during pregnancy and child neuropsychological development in the INMA-Sabadell cohort.
It is suggested that prenatal BPA exposure does not affect cognitive development up to age 4 years, and associations are observed with psychomotor development and ADHD-related symptoms at early ages, but these do not appear to persist until later ages.
Associations of Prenatal Urinary Bisphenol A Concentrations with Child Behaviors and Cognitive Abilities
Prenatal urinary BPA concentration was associated with some aspects of child behavior in this cohort, and some associations were stronger among boys than among girls, and this association was not modified by sex.
Impact of Early-Life Bisphenol A Exposure on Behavior and Executive Function in Children
G gestational BPA exposure affected behavioral and emotional regulation domains at 3 years of age, especially among girls, and Clinicians may advise concerned patients to reduce their exposure to certain consumer products, but the benefits of such reductions are unclear.
Prenatal phenolic compounds exposure and neurobehavioral development at 2 and 7years of age.
Prenatal exposure to BPA affects neurocognitive development, and this effect differs between 7-year-old boys and girls, and more studies are needed to elucidate the relationship between phenolic compound exposure in utero and children's neurobehavioral development.
Early life bisphenol A exposure and neurobehavior at 8years of age: Identifying windows of heightened vulnerability.
It is found that sex-dependent associations between BPA and child neurobehavior may depend on the timing of BPA exposure.
Prenatal and early childhood bisphenol A concentrations and behavior in school-aged children.
This study adds to the existing literature showing associations of early life BPA exposure with behavior problems, including anxiety, depression, and hyperactivity in children, by measuring BPA in urine from mothers during pregnancy and children at 5 years of age.
Bisphenol A exposure and symptoms of anxiety and depression among inner city children at 10-12 years of age.
Evidence is provided that prenatal BPA exposure is associated with more symptoms of anxiety and depression in boys but not in girls at age 10-12 years, compared with other studies investigating gestational exposure to Bisphenol A.
Early prenatal exposure to suspected endocrine disruptor mixtures is associated with lower IQ at age seven.
Among boys, early prenatal exposure to EDCs was associated with lower intellectual functioning at age 7 and bisphenol F was identified as the primary chemical of concern, suggesting that the BPA replacement compound may not be any safer for children.
Cord blood BPA level and child neurodevelopment and behavioral problems: The Hokkaido Study on Environment and Children's Health.
The findings suggested no association between cord blood BPA level and child mental, psychomotor and behavioral problems at different ages in association with prenatal BPA exposures in the prospective birth cohort study, however, cord blood bisphenol A level may be associated with child Behavioral problems at early age.