Prenatal Exposure to Lead, δ-Aminolevulinic Acid, and Schizophrenia: Further Evidence

@inproceedings{Opler2008PrenatalET,
  title={Prenatal Exposure to Lead, δ-Aminolevulinic Acid, and Schizophrenia: Further Evidence},
  author={Mark Opler and Stephen L. Buka and Justina L Groeger and Ian W. McKeague and Catherine Wei and Pam Factor-Litvak and Michaeline A Bresnahan and Joseph H. Graziano and Jill M. Goldstein and Larry J. Seidman and A. Sloman Brown and Ezra Saul Susser},
  booktitle={Environmental health perspectives},
  year={2008}
}
BACKGROUND A previously conducted study of prenatal lead exposure and schizophrenia using delta-aminolevulinic acid, a biologic marker of Pb exposure, in archived maternal serum samples collected from subjects enrolled in the Childhood Health and Development Study (1959-1966) based in Oakland, California, suggested a possible association between prenatal Pb exposure and the development of schizophrenia in later life. OBJECTIVES In the present study we extend these findings using samples… CONTINUE READING
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