Potentiation of nitric oxide-mediated vasorelaxation by xanthine oxidase inhibitors.

Yusuke Miyamoto, T. Akaike, M. Yoshida, S. Goto, Hiromichi Horie, H. Maeda
Published 1996 in
Proceedings of the Society for Experimental…

Abstract

Nitric oxide (NO), now almost synonymous with endothelium-derived relaxing factor (EDRF), reacts with superoxide anion radical (O2-) and forms a potentially toxic molecular species, peroxynitrite (ONOO-). Because xanthine oxidase (XO) seems to be a major O2- -producing enzyme in the vascular system, it is important to clarify the mechanism of XO regulation… (More)

Therapeutic potential of pegylated hemin for reactive oxygen species-related diseases via induction of heme oxygenase-1: results from a rat hepatic ischemia/reperfusion injury model.

Jun Fang, Haibo Qin, +4 authors Hiroshi Maeda
The Journal of pharmacology and experimental…
2011

Potentiation of nitric oxide-mediated vasorelaxation by xanthine oxidase inhibitors.

Abstract

Topics

Figures and Tables

Cited By

SMA-copolymer conjugate of AHPP: a polymeric inhibitor of xanthine oxidase with potential antihypertensive effect.

Therapeutic potential of pegylated hemin for reactive oxygen species-related diseases via induction of heme oxygenase-1: results from a rat hepatic ischemia/reperfusion injury model.

Carbon monoxide releasing molecule induces endothelial nitric oxide synthase activation through a calcium and phosphatidylinositol 3-kinase/Akt mechanism.

Endothelin-1 stimulates catalase activity through the PKCδ-mediated phosphorylation of serine 167.

Manipulating Redox Signaling to Block Tumor Angiogenesis

Use of uric acid-lowering agents limits experimental cyclosporine nephropathy.

Therapeutic effects of xanthine oxidase inhibitors: renaissance half a century after the discovery of allopurinol.

Carbon monoxide regulates blood pressure cooperatively with nitric oxide in hypertensive rats

Endothelium-derived hyperpolarizing factor synthase (Cytochrome P450 2C9) is a functionally significant source of reactive oxygen species in coronary arteries.

Effects of the flavoprotein inhibitor, diphenyleneiodonium sulfate, on ex vivo organic nitrate tolerance in the rat.

References

Slides referencing similar topics

Potentiation of nitric oxide-mediated vasorelaxation by xanthine oxidase inhibitors.

Abstract

Topics

Figures and Tables

Cited By

SMA-copolymer conjugate of AHPP: a polymeric inhibitor of xanthine oxidase with potential antihypertensive effect.

Therapeutic potential of pegylated hemin for reactive oxygen species-related diseases via induction of heme oxygenase-1: results from a rat hepatic ischemia/reperfusion injury model.

Carbon monoxide releasing molecule induces endothelial nitric oxide synthase activation through a calcium and phosphatidylinositol 3-kinase/Akt mechanism.

Endothelin-1 stimulates catalase activity through the PKCδ-mediated phosphorylation of serine 167.

Manipulating Redox Signaling to Block Tumor Angiogenesis

Use of uric acid-lowering agents limits experimental cyclosporine nephropathy.

Therapeutic effects of xanthine oxidase inhibitors: renaissance half a century after the discovery of allopurinol.

Carbon monoxide regulates blood pressure cooperatively with nitric oxide in hypertensive rats

Endothelium-derived hyperpolarizing factor synthase (Cytochrome P450 2C9) is a functionally significant source of reactive oxygen species in coronary arteries.

Effects of the flavoprotein inhibitor, diphenyleneiodonium sulfate, on ex vivo organic nitrate tolerance in the rat.

References

Similar Papers

Slides referencing similar topics