Postprandial alterations in hemodynamics and blood pressure in normal subjects.

@article{Fagan1986PostprandialAI,
  title={Postprandial alterations in hemodynamics and blood pressure in normal subjects.},
  author={Timothy C. Fagan and Phyllis R. Sawyer and L A Gourley and J T Lee and Thomas E. Gaffney},
  journal={The American journal of cardiology},
  year={1986},
  volume={58 7},
  pages={
          636-41
        }
}
Time course and nature of postprandial haemodynamic changes in normal man.
TLDR
Eating itself did not alter venous plasma noradrenaline, and plasma adrenaline even tended to decrease, which reflects both the roughness of venous catecholamines in estimating adrenergic changes and the complexity of the underlying mechanisms and related reflexes.
Postprandial changes in supine and erect heart rate, systemic blood pressure and plasma noradrenaline and renin activity in normal subjects
TLDR
A food-induced vasodepressor response combined with baroreceptor resetting is considered to have occurred in this population of normal fasting subjects, and eating should be considered as an important potential source of bias in cardiovascular studies.
The effect of meal size on the cardiovascular responses to food ingestion
TLDR
There are substantial peripheral and central cardiovascular changes after food in man and there appears to be a relationship between meal size and the extent of these changes.
Relationship Between Age and Response to Meals the Cardiovascular
TLDR
The greater blood pressure reduction after meals in older patients may be due to decreased baroreflex sensitivity in association with higher arterial pressures, and the changes in blood pressure due to meals may confound the diagnosis of hypertension and interfere with the interpretation of the response to antihypertensive treatment.
Relationship between age and the cardiovascular response to meals
TLDR
The greater blood pressure reduction after meals in older patients may be due to decreased baroreflex sensitivity in association with higher arterial pressures, and the changes in blood pressure due to meals may confound the diagnosis of hypertension and interfere with the interpretation of the response to antihypertensive treatment.
Relationship between postprandial changes in cardiac left ventricular function, glucose and insulin concentrations, gastric emptying, and satiety in healthy subjects
TLDR
This study shows that postprandial CO, HR, SV and LV longitudinal systolic and diastolic functions increase concomitantly with increased satiety, antral area, and glucose and insulin levels.
Hemodynamic and Autonomic Nervous System Responses to Mixed Meal Ingestion in Healthy Young and Old Subjects and Dysautonomic Patients With Postprandial Hypotension
TLDR
Power spectral analysis suggests an impairment in the postprandial autonomic modulation of HR in healthy elderly and dysautonomic subjects, possibly predisposing to hypotension when vascular compensation is inadequate.
Cardiovascular and hormonal responses to a meal in hypertrophic cardiomyopathy: A comparison of patients with and without postprandial exacerbation of symptoms
TLDR
There is no evidence for a distinctive hemodynamic or hormonal response to food in hypertrophic cardiomyopathy patients with postprandial symptoms, and these symptoms more likely reflect differences in underlying cardiac disease characteristics and severity.
Postprandial ambulatory blood pressure and heart rate effects in healthy elderly adults.
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References

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Effects of meals on hemodynamics: Implications for antihypertensive drug studies
TLDR
The ingestion of food is known to affect blood pressure and heart rate, but food is often allowed in patients under observation for antihypertensive drug effects and should be avoided whenever possible.
Postprandial reduction in blood pressure in the elderly.
TLDR
Observations show that postprandial reductions in blood pressure may predispose the elderly to symptomatic hypotension andcompensatory cardioacceleration was minimal in the elderly, suggesting impaired baroreflexes.
Systemic and Regional Hemodynamic Changes during Food Intake and Digestion in Nonanesthetized Dogs
TLDR
Results indicate that a redistribution of blood flow occurs during digestion with a preference for the vascular bed of the superior mesenteric artery.
Hemodynamic and humoral effects of caffeine in autonomic failure. Therapeutic implications for postprandial hypotension.
TLDR
It is concluded that caffeine is a pressor agent and attenuates postprandial hypotension in autonomic failure, and that this effect is not primarily due to elevations in sympathoadrenal activity or activation of the renin-angiotensin system.
Age-related changes of baroreflex function, plasma norepinephrine, and blood pressure.
TLDR
An increase in plasma norepinephrine levels with age could be mediated by the age-related change of baroreflex sensitivity, which could be causally related to the elevation of blood pressure.
Glucose and baroreceptor function. Effects of oral administration of glucose on baroreceptor function in cerebrovascular disease and in other disorders with baroreceptor reflex block.
TLDR
Some patients with diabetes, with or without clinically recognizable peripheral neuropathy, have impaired baroreceptor reflexes, and in such subjects further impairment of vasomotor function related to the meal may significantly decrease cerebral blood flow in a compromised part of the vasculature.
Coronary and visceral vasoactivity associated with eating and digestion in the conscious dog.
TLDR
Modifications in regional blood flow and vasoactivity in response to eating and digestion were studied in conscious dogs after full recovery from instrumentation with ultrasonic or electromagnetic flow probes on the ascending aorta, left circumflex coronary, mesenteric, renal and iliac arteries, and miniature pressure gauges in the aortA.
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