Polycystic ovary syndrome: a simplified approach based on the evolving set of symptoms.

@article{Orvieto2003PolycysticOS,
  title={Polycystic ovary syndrome: a simplified approach based on the evolving set of symptoms.},
  author={Raoul Orvieto and Zion Ben-Rafael},
  journal={Human reproduction},
  year={2003},
  volume={18 4},
  pages={
          896-7
        }
}
similar regardless of whether urinary or recombinant gonadotrophins are used. In this article the author refers to the study of Gleicher et al. (2000), which makes no comparison between urinary and recombinant products, but instead questions whether ovulation induction in general should be replaced by IVF as a means of reducing the risk of higher order multiple pregnancy. To conclude, I feel that if we are to make progress in terms of how best to manage patients undergoing ART then we need to… 
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TLDR
Several studies have examined the genetic basis of polycystic ovary syndrome with varying results, probably caused by the different criteria used to define the syndrome.
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This debate wishes to re-explore the current thinking on PCOS, with a particular emphasis on the British and European perspective and invite others to contribute to the discussion which could form the basis for an international consensus.
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A consensus for a unifying balanced and practical working definition of polycystic ovarian syndrome is proposed, which if universally adopted as a standard could contribute much to all future work involving this most prevalent of syndromes.
Polycystic ovary syndrome
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This Primer summarizes the current state of knowledge regarding the epidemiology, mechanisms and pathophysiology, diagnosis, screening and prevention, management and future investigational directions of the disorder.
Ovulation after glucocorticoid suppression of adrenal androgens in the polycystic ovary syndrome is not predicted by the basal dehydroepiandrosterone sulfate level.
TLDR
The data from this prospective study do not suggest that continuous dexamethasone suppression results in consistent ovulation in any PCOS patient, regardless of basal DHEAS levels, and suggest that AA may be an important risk factor for PCOS, once the syndrome is established, they play a limited role in the associated ovulatory dysfunction.
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TLDR
Exposure to hypoglycemia as a model of stress was not followed by hypersecretion of adrenal androgens and revealed no signs of steroid enzyme disturbances in women with PCOS, suggesting an adaptation to increased adrenal reactivity to endogenous ACTH in womenWith PCOS.
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