Plasmodium falciparum-infected erythrocytes induce NF-kappaB regulated inflammatory pathways in human cerebral endothelium.

@article{Tripathi2009PlasmodiumFE,
  title={Plasmodium falciparum-infected erythrocytes induce NF-kappaB regulated inflammatory pathways in human cerebral endothelium.},
  author={A. Tripathi and W. Sha and V. Shulaev and M. Stins and D. Sullivan},
  journal={Blood},
  year={2009},
  volume={114 19},
  pages={
          4243-52
        }
}
Cerebral malaria is a severe multifactorial condition associated with the interaction of high numbers of infected erythrocytes to human brain endothelium without invasion into the brain. The result is coma and seizures with death in more than 20% of cases. Because the brain endothelium is at the interface of these processes, we investigated the global gene responses of human brain endothelium after the interaction with Plasmodium falciparum-infected erythrocytes with either high- or low-binding… Expand
Plasmodium falciparum histones induce endothelial proinflammatory response and barrier dysfunction.
Association of Plasmodium falciparum with Human Endothelial Cells in vitro
Cytokines and Chemokines in Cerebral Malaria Pathogenesis
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