Penicillin-G induced interictal activity increases both opioid peptide tissue content and in vitro release in the rat brain

@article{Asai1995PenicillinGII,
  title={Penicillin-G induced interictal activity increases both opioid peptide tissue content and in vitro release in the rat brain},
  author={Miguel Asai and A. Mart{\'i}nez and Rodrigo Fern{\'a}ndez-Mas and Augusto Fern{\'a}ndez-Guardiola},
  journal={Neuropeptides},
  year={1995},
  volume={29},
  pages={163-170}
}
8 Citations

Long-term pharmacological kindling increases in vitro release of IR-Met and IR-Leu-enkephalin from amygdala.

EFECTO DE LA NALOXONA SOBRE UN FOCO EPILÉPTICO INDUCIDO POR PENICILINA EN LA AMÍGDALA DEL LÓBULO TEMPORAL DE GATOS. EEG Y REGISTROS POLISOMNOGRÁFICOS DE 23 HORAS

Naloxone administration blocks the inhibitory effect of the opioids (liberated in the amygdala by the application of Pn) on the onset of the REM sleep and an activation of the mechanisms of induction of the SWS-II, points out to the possibility that enkephalinergic inhibitory processes control the appearance of theREM sleep.

Endogenous opiates: 1995

MAPEO CORTICAL QUE MUESTRA EL EFECTO DE LAS ENCEFALINAS EN UN FOCO EPILÉPTICO

Introduccion: La actividad epileptica modifica el sistema de los opioides endogenos al aumentar su concentracion al termino de la fase octal, en la fase postictal y durante la fase interictal. Este

EFECTO DE LA ESTIMULACIÓN DEL NERVIO VAGO SOBRE LA EPILEPSIA FOCAL AMIGDALINA EN LA RATA

Antecedentes: La estimulacion del nervio vago (ENV) es una terapia anticonvulsiva que se usa en la epilepsia resistente al tratamiento farmacologico. Para encontrar el mecanismo de accion del efecto

References

SHOWING 1-10 OF 49 REFERENCES

Met-enkephalin release from slices of the rat striatum and globus pallidus: stimulation by excitatory amino acids.

The present study evaluated the effects of high K+ and four excitatory amino acids (EAAs) on the release of met-enkephalin-like immunoreactivity from slices of the rat striatum and globus pallidus, finding that the activation of NMDA receptors by an endogenous agonist did not contribute to the enhancement.