Parvalbumin immunoreactivity and expression of GABAA receptor subunits in the thalamus after experimental TBI.

@article{Huusko2014ParvalbuminIA,
  title={Parvalbumin immunoreactivity and expression of GABAA receptor subunits in the thalamus after experimental TBI.},
  author={Noora Huusko and Asla Pitk{\"a}nen},
  journal={Neuroscience},
  year={2014},
  volume={267},
  pages={30-45}
}
Traumatic brain injury (TBI) causes 10-20% of acquired epilepsy in humans, resulting in an ictogenic region that is often located in the cerebral cortex. The thalamus provides heavy projections to the cortex and the activity of thalamocortical pathways is controlled by GABAergic afferents from the reticular nucleus of the thalamus (RT). As rats with TBI induced by lateral fluid-percussion injury (FPI) undergo epileptogenesis, we hypothesized that damage to the parvalbumin (PARV)-immunoreactive… CONTINUE READING

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