Oxidized LDLs trigger endoplasmic reticulum stress and autophagy: prevention by HDLs.

Abstract

Oxidized LDLs (oxLDLs) induce various cellular dysfunctions potentially implicated in the pathogenesis of atherosclerosis. For instance, toxic concentrations of oxLDLs trigger ER stress, autophagy and apoptosis. High-density lipoproteins (HDLs) counteract several adverse biological effects triggered by oxLDLs. Our recent study reveals that HDLs inhibit the activation of ER stress and of autophagy induced by oxLDLs.

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@article{Muller2011OxidizedLT, title={Oxidized LDLs trigger endoplasmic reticulum stress and autophagy: prevention by HDLs.}, author={Carole Muller and Robert Salvayre and Anne N{\`e}gre-Salvayre and C{\'e}cile Vindis}, journal={Autophagy}, year={2011}, volume={7 5}, pages={541-3} }