Oscillopsia: causes and management.

  title={Oscillopsia: causes and management.},
  author={Caroline Tilikete and A. Vighetto},
  journal={Current opinion in neurology},
  volume={24 1},
PURPOSE OF REVIEW Oscillopsia is an illusion of an unstable visual world. It is associated with poor visual acuity and is a disabling and distressing condition reported by numerous patients with neurological disorders. The goal of this study is to review the recent findings in the various pathophysiological mechanisms of oscillopsia and the potential treatments available. RECENT FINDINGS Oscillopsia most often results from abnormal eye movements or from impaired vestibulo-ocular reflex. A… 
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[Oscillopsia: pathophysiological mechanisms and treatment].
The physiology of the systems subserving stable vision, the various pathophysiological mechanisms of oscillopsia, and the different treatments available are reviewed to suggest that some visuo-perceptive deficits consecutive to temporo-parietal lesions resemble oscillosia and could result from a deficit in elaborating spatial constancy.
Deficient high-acceleration vestibular function in patients with polyneuropathy
The finding that two thirds of patients with axonal or demyelinating polyneuropathy showed unilateral or bilateral gain reductions of the horizontal high-acceleration vestibulo-ocular reflex suggests that, in many patients with PNP, the neuropathic process includes the vestibular nerve.
Superior oblique myokymia: efficacy of medical treatment.
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This is the first study to identify abnormalities of fixation in MND and results indicate that ocular fixation instabilities may be a marker of the sub-clinical frontal lobe dysfunction in M ND.
Gaze fixation deficits and their implication in ataxia–telangiectasia
Degeneration of cerebellar Purkinje neurons disinhibit the caudal fastigial oculomotor region (FOR) and vestibular nuclei (VN), which can cause nystagmus, including PAN, while disinhibition of FOR can affect saccade generating mechanisms, leading to SWSI and SO.
Measurement of dynamic visual acuity in patients with vestibular areflexia
The test is simple and sensitive enough to separate normal subjects from patients suffering from a vestibular loss and there was a good correlation between the objective results and the subjective complaint of oscillopsia.
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Spinocerebellar ataxia type 6 (SCA6) is a calcium channelopathy due to a pathological CAG repeat expansion in CACNL1A4. Patients frequently describe paroxysmal vertigo early in the disease course,
Upbeat nystagmus from a demyelinating lesion in the caudal pons.
Clinicoanatomic correlation in this patient suggests that a lesion of the superior vestibular nucleus and its efferent crossing ventral tegmental tract could be responsible for the primary position upbeat nystagmus.
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Superior canal dehiscence syndrome, or Minor’s syndrome, is a recently described disease that results from the absence of bone over the superior semicircular canal which results in the communication
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It is shown that ocular fixation is impaired in patients with parkinsonian conditions and may prove useful as part of an oculomotor profile to aid with the differentiation of parkinsonians conditions.