Nolz1 is induced by retinoid signals and controls motoneuron subtype identity through distinct repressor activities.


The acquisition and maintenance of final neuronal identity depends in part upon the implementation of fate-specification programs in postmitotic neurons; however, the mechanisms involved remain unclear. In the developing spinal cord, retinoic acid (RA) signaling pathways specify the columnar and divisional identities of postmitotic motoneurons (MNs). Here… (More)
DOI: 10.1242/dev.028043


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