No neurochemical evidence of brain injury after blast overpressure by repeated explosions or firing heavy weapons

@article{Blennow2011NoNE,
  title={No neurochemical evidence of brain injury after blast overpressure by repeated explosions or firing heavy weapons},
  author={Kaj Blennow and Michael A G Jonsson and Neils Andreasen and Lars E. Rosengren and Anders Wallin and P. A. Hellstr{\"o}m and Henrik Zetterberg},
  journal={Acta Neurologica Scandinavica},
  year={2011},
  volume={123}
}
Blennow K, Jonsson M, Andreasen N, Rosengren L, Wallin A, Hellström PA, Zetterberg H. No neurochemical evidence of brain injury after blast overpressure by repeated explosions or firing heavy weapons.Acta Neurol Scand: 2011: 123: 245–251.© 2010 John Wiley & Sons A/S. 
Brain injuries from blasts.
  • A. Ropper
  • Medicine
    The New England journal of medicine
  • 2011
For soldiers wounded in the wars in Iraq and Afghanistan, a disproportionate number of injuries are from blasts resulting from the numerous improvised explosive devices deployed by insurgent forces.
Neurological Effects of Repeated Exposure to Military Occupational Levels of Blast: A Review of Scientific Literature
There is increasing concern over the common, repetitive forms of blast—often unrelated to combat—to which military members are exposed during service, and how those exposures could affect the central
Molecular Aspects of Concussion and Chronic Traumatic Encephalopathy
TLDR
Chronic traumatic encephalopathy (CTE) is a progressive, neurodegenerative disease caused by repetitive mild traumatic brain injury(mTBI).
Blast-related traumatic brain injury
TLDR
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Assessing Neuro-Systemic & Behavioral Components in the Pathophysiology of Blast-Related Brain Injury
TLDR
An overview of the current status of the cellular and pathological mechanisms involved in blast overpressure injury is provided and it is argued for the urgent need to identify potential biomarkers that can hint at the different mechanisms involved.
Repeated primary blast injury causes delayed recovery, but not additive disruption, in an in vitro blood-brain barrier model.
TLDR
Careful investigation of the effects of repeated blast on the BBB will help identify injury levels and a temporal window of vulnerability associated with BBB dysfunction, ultimately leading to improved strategies for protecting warfighters against repeated blast-induced disruption of the cerebral vasculature.
Myelin Water Fraction Is Transiently Reduced after a Single Mild Traumatic Brain Injury – A Prospective Cohort Study in Collegiate Hockey Players
TLDR
Myelin disruption was not apparent in the athletes who did not experience a concussion, despite exposure to repetitive subconcussive trauma over a season of collegiate hockey, suggesting transient myelin disruption following a single mTBI, with subsequent remyelination of affected neurons.
Terahertz spectroscopic diagnosis of early blast-induced traumatic brain injury in rats.
TLDR
It is suggested that this method has potential as an alternative method for high-sensitive, rapid, label-free, economical and early diagnosis of bTBI.
Tightly coupled repetitive blast-induced traumatic brain injury: development and characterization in mice.
TLDR
The data suggest that repeated blast exposures lead to increased impairment severity in multiple neurological parameters of TBI in mice.
Computational Study on the Bridging Vein Rupture of Blast-Induced Traumatic Brain Injury Using a Numerical Human Head Model
The occurrence of blast-induced traumatic brain injury (bTBI) in people serving in battle environments is dramatically high. The blast front, or leading edge of the shock wave is a brief, acute
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