Nitrous Oxide and Xenon Prevent Amphetamine-Induced Carrier-Mediated Dopamine Release in a Memantine-Like Fashion and Protect Against Behavioral Sensitization

@article{David2006NitrousOA,
  title={Nitrous Oxide and Xenon Prevent Amphetamine-Induced Carrier-Mediated Dopamine Release in a Memantine-Like Fashion and Protect Against Behavioral Sensitization},
  author={H{\'e}l{\`e}ne N. David and Marc Ansseau and Marc Lemaire and Jacques H Abraini},
  journal={Biological Psychiatry},
  year={2006},
  volume={60},
  pages={49-57}
}

Argon blocks the expression of locomotor sensitization to amphetamine through antagonism at the vesicular monoamine transporter-2 and mu-opioid receptor in the nucleus accumbens

We investigated the effects of the noble gas argon on the expression of locomotor sensitization to amphetamine and amphetamine-induced changes in dopamine release and mu-opioid neurotransmission in

Argon prevents the development of locomotor sensitization to amphetamine and amphetamine-induced changes in mu opioid receptor in the nucleus accumbens

It is indicated that argon inhibits the development of both locomotor sensitization and mu opioid receptor activation induced by repeated administration of amphetamine.

Short‐term development of behavioral sensitization to amphetamine requires N‐methyl‐D‐aspartate‐ and nicotinic‐dependent mechanisms in the nucleus accumbens

It is reported that the concomitant blockade of glutamatergic and nicotinic ionotropic receptors in the core of the nucleus accumbens blocks the development of behavioral sensitization to amphetamine and further abolishes the increase in extracellular dopamine release induced by amphetamine in the nucleus Accumbens.

The effect of sertindole on behavioural sensitisation to methamphetamine in mice

The present study assessed whether memantine would influence behavioural sensitisation to the stimulatory effects of methampheta- mine on mouse locomotion.

Mechanism of block and behavioral effects of the N-methyl-D-aspartate receptor antagonists memantine and ketamine

The research shows NMDA receptors possess a second binding site at which memantine, but not ketamine, can inhibit activity, and the dramatic effect physiological concentrations of magnesium has on the ability of these drugs to inhibit NMDA receptor activity.

In vivo evidence for functional NMDA receptor blockade by memantine in rat hippocampal neurons

Therapeutic doses of memantine are able to antagonize NMDA receptor-mediated activity in the principal cells of the hippocampus in vivo, i.e. in the presence of physiological concentrations of Mg2+.

Implication of NMDA receptors in behavioural sensitization to psychostimulants: a short review.

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