Nitric oxide synthesis inhibition does not impair water immersion-induced renal vasodilation in humans.

@article{DijkhorstOei2000NitricOS,
  title={Nitric oxide synthesis inhibition does not impair water immersion-induced renal vasodilation in humans.},
  author={Lioe-Ting Dijkhorst-Oei and P. C. den Boer and Ton J. Rabelink and Hein A. Koomans},
  journal={Journal of the American Society of Nephrology : JASN},
  year={2000},
  volume={11 7},
  pages={
          1293-302
        }
}
Nitric oxide (NO) is tonically released in the kidney to maintain renal perfusion and adequate sodium and water clearance. Little is known about the role of NO in the renal adaptation to an acute volume challenge. This is important for our understanding of pathophysiologic conditions associated with impaired NO activity. This study examined the effects of NO synthesis inhibition on neurohumoral, renal hemodynamic, and excretory responses to head-out immersion (HOI). Seven healthy men underwent… CONTINUE READING

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