Nitric oxide induces neurotransmitter release from hippocampal slices.

@article{Lonart1992NitricOI,
  title={Nitric oxide induces neurotransmitter release from hippocampal slices.},
  author={Gy{\"o}rgy Lonart and J. Wang and K. M. Johnson},
  journal={European journal of pharmacology},
  year={1992},
  volume={220 2-3},
  pages={
          271-2
        }
}

Nitric oxide induces calcium‐dependent release of [3H]dopamine release from striatal slices

TLDR
The data suggest that NO induces calcium‐dependent [3H]DA release from the striatum via a mechanism which is independent of blockade of electron transport or activation of guanylate cyclase, as well as the possibility that NO acted through blockade of mitochondrial electron transport.

Role of nitric oxide in modulating neurotransmitter release from rat striatum

Effect of nitric oxide donors on endogenous dopamine release from rat striatal slices. II : The role of voltage-dependent sodium channels, calcium channel activation, reverse transport mechanism, guanylate cyclase and endogenous glutamate

TLDR
Results presented here indicate that NO-donors SNP and HA stimulate DA release by facilitating reverse DA transport and that neither endogenous glutamate nor guanylate cyclase activation plays an intermediary role in stimulatory effects of NO- donors on DA release from rat striatal slices.

Release of Adenosine from Rat Hippocampal Slices by Nitric Oxide Donors

TLDR
Direct evidence that NO evokes a concentration‐dependent release of adenosine from both stimulated and unstimulated nerves of the hippocampus is provided, supporting the proposal that the effects of SNAP were due to the liberation of NO.
...

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