Nitric oxide alters arachidonic acid turnover in brain cortex synaptoneurosomes

@article{Chalimoniuk2006NitricOA,
  title={Nitric oxide alters arachidonic acid turnover in brain cortex synaptoneurosomes},
  author={M. Chalimoniuk and J. Głowacka and A. Zabielna and A. Eckert and J. Strosznajder},
  journal={Neurochemistry International},
  year={2006},
  volume={48},
  pages={1-8}
}
Nitric oxide (NO) and arachidonic acid (AA) and also its metabolites are very important inter- and intracellular second messengers. They are involved in mechanisms of learning and memory. However, liberated in excessive amount in brain ischemia, Parkinson and Alzheimer diseases they are responsible for cell degeneration and death. Previously, we could show that Alzheimer disease's amyloid-beta protein enhanced nitric oxide liberation. The role of NO in AA metabolism is till now not well… Expand
Molecular mechanism of PC12 cell death evoked by sodium nitroprusside, a nitric oxide donor.
Alpha-synuclein decreases arachidonic acid incorporation into rat striatal synaptoneurosomes.
Effect of nitric oxide donors on membrane tritium accumulation of endocannabinoids and related endogenous lipids.
Nitric oxide modulation of the basal ganglia circuitry: therapeutic implication for Parkinson's disease and other motor disorders.
...
1
2
3
...

References

SHOWING 1-10 OF 54 REFERENCES
The neuromessenger platelet-activating factor in plasticity and neurodegeneration.
  • N. Bazan
  • Biology, Medicine
  • Progress in brain research
  • 1998
Nitric oxide as modulator of neuronal function
Phospholipase A2 and Its Role in Brain Tissue
...
1
2
3
4
5
...