New aspects in the pathophysiology of rapid eye movement sleep behavior disorder: the potential role of glutamate, gamma-aminobutyric acid, and glycine.

@article{Luppi2013NewAI,
  title={New aspects in the pathophysiology of rapid eye movement sleep behavior disorder: the potential role of glutamate, gamma-aminobutyric acid, and glycine.},
  author={Pierre-Herv{\'e} Luppi and Olivier Cl{\'e}ment and Sara Valencia Garcia and Fr{\'e}d{\'e}ric Brischoux and Patrice Fort},
  journal={Sleep medicine},
  year={2013},
  volume={14 8},
  pages={714-8}
}
Rapid eye movement sleep behavior disorder (RBD) is a parasomnia characterized by the occurrence of intense movements during rapid eye movement (REM) sleep, also named paradoxical sleep. The neuronal dysfunctions at the origin of the loss of atonia in RBD patients are not known. One possibility is that RBD is due to the degeneration of neurons inducing the muscle atonia of REM sleep. Therefore, in our paper we review data on the populations of neurons responsible for the atonia of REM sleep… CONTINUE READING

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