New Thinking on the Etiology and Pathogenesis of Late-Onset Alzheimer's Disease

Abstract

For many years, it has been accepted that the etiology of early-onset Alzheimer's disease (AD) is genetic in nature, with the relevant mutations involving genes encoding products that function in pathways dealing with processing of β-amyloid. The etiology of the far more prevalent late-onset AD is not primarily genetic in origin even though research over… (More)
DOI: 10.4061/2011/848395

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