Neuronal to oligodendroglial α-synuclein redistribution in a double transgenic model of multiple system atrophy.

@article{Rockenstein2012NeuronalTO,
  title={Neuronal to oligodendroglial α-synuclein redistribution in a double transgenic model of multiple system atrophy.},
  author={Edward M. Rockenstein and Kiren K. Ubhi and Chandra A. Inglis and Michael Mante and Christina Patrick and Anthony Adame and Eliezer Masliah},
  journal={Neuroreport},
  year={2012},
  volume={23 4},
  pages={259-64}
}
Multiple system atrophy is a sporadic, progressive, neurodegenerative disease characterized by an oligodendroglial accumulation of alpha-synuclein (α-syn). The mechanisms underlying the oligodendroglial accumulation of α-syn in the brains of patients with multiple system atrophy have attracted a great deal of interest, given the primarily neuronal role reported for this protein. We examined the interactions between neuronal and oligodendroglial α-syn in the progeny of crosses between parental… CONTINUE READING
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