Neuronal activity determined by quantitative EEG and cortical microdialysis is increased following controlled cortical impact injury in rats.

Abstract

Following brain injury increased glutamate release is linked to sustained neuronal activation resulting in excitotoxic tissue damage. Isoflurane anesthesia has been shown to decrease electroencephalographic (EEG) activity and extracellular (e.c.) glutamate, possibly attenuating excitotoxic tissue damage. However, based on clinical experience EEG activity… (More)

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