Neurochemical changes after morphine, dizocilpine or riluzole in the ventral posterolateral thalamic nuclei of rats with hyperalgesia.

@article{Abarca2000NeurochemicalCA,
  title={Neurochemical changes after morphine, dizocilpine or riluzole in the ventral posterolateral thalamic nuclei of rats with hyperalgesia.},
  author={Carolina Abarca and Ewelyn Silva and M. J. Sepulveda and P Oliva and Enrique Contreras},
  journal={European journal of pharmacology},
  year={2000},
  volume={403 1-2},
  pages={
          67-74
        }
}
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28 Citations
Highly Influential Citations
2
Background Citations
16
Methods Citations
2
Results Citations
2

28 Citations

The NMDA antagonist MK-801 induces hyperalgesia and increases CSF excitatory amino acids in rats: Reversal by guanosine

Riluzole rapidly attenuates hyperemotional responses in olfactory bulbectomized rats, an animal model of depression

Extracellular glutamate, aspartate and arginine increase in the ventral posterolateral thalamic nucleus during nociceptive stimulation

Antinociceptive effect of riluzole in rats with neuropathic spinal cord injury pain.

Riluzole may be an effective clinical analgesic for the treatment of below-level neuropathic SCI pain and there is a predominant supraspinal component of riluzoles-induced antinociception in SCI rats.

The effects of morphine on basal neuronal activities in the lateral and medial pain pathways

Riluzole produces distinct anxiolytic-like effects in rats without the adverse effects associated with benzodiazepines

Ablation of IB4 non-peptidergic afferents in the rat facet joint prevents injury-induced pain and thalamic hyperexcitability via supraspinal glutamate transporters

blation of IB 4 non-peptidergic afferents in the rat facet joint revents injury-induced pain and thalamic hyperexcitability via upraspinal glutamate transporters

It is suggested that a painful facet injury induces changes extending to supraspinal structures and that IB4-positive afferents in the facet joint may be critical for the development and maintenance of sensitization in the thalamus after a painfulacet joint injury.

Selective downregulation of vesicular glutamate transporter2 in ventral posterolateral nucleus of thalamus attenuates neuropathic mechanical allodynia in mice

Gi protein functions in thalamic neurons to decrease orofacial nociceptive response

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