Neurochemical changes after morphine, dizocilpine or riluzole in the ventral posterolateral thalamic nuclei of rats with hyperalgesia.

@article{Abarca2000NeurochemicalCA,
  title={Neurochemical changes after morphine, dizocilpine or riluzole in the ventral posterolateral thalamic nuclei of rats with hyperalgesia.},
  author={Carolina Abarca and E Silva and M. J. Sepulveda and P Oliva and Enrique Contreras},
  journal={European journal of pharmacology},
  year={2000},
  volume={403 1-2},
  pages={
          67-74
        }
}
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28 Citations
Highly Influential Citations
2
Background Citations
15
Methods Citations
2
Results Citations
2

28 Citations

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Riluzole may be an effective clinical analgesic for the treatment of below-level neuropathic SCI pain and there is a predominant supraspinal component of riluzoles-induced antinociception in SCI rats.

The effects of morphine on basal neuronal activities in the lateral and medial pain pathways

Estradiol attenuates spinal cord injury-related central pain by decreasing glutamate levels in thalamic VPL nucleus in male rats

Results suggest that glutamate in the VPL nucleus of the thalamus is involved in the pathology of neuropathic pain after STT injury; furthermore, 17β-estradiol may attenuate this neuropathicPain by decreasing glutamate levels.

Riluzole produces distinct anxiolytic-like effects in rats without the adverse effects associated with benzodiazepines

Ablation of IB4 non-peptidergic afferents in the rat facet joint prevents injury-induced pain and thalamic hyperexcitability via supraspinal glutamate transporters

Ethanol-induced alterations of amino acids measured by in vivo microdialysis in rats: a meta-analysis

The results provide standardized basal values for future experimental and in silico studies on neurotransmitters release in the rat brain and may be helpful to understand the effect of ethanol on neurotransmitter release.

blation of IB 4 non-peptidergic afferents in the rat facet joint revents injury-induced pain and thalamic hyperexcitability via upraspinal glutamate transporters

It is suggested that a painful facet injury induces changes extending to supraspinal structures and that IB4-positive afferents in the facet joint may be critical for the development and maintenance of sensitization in the thalamus after a painfulacet joint injury.

Selective downregulation of vesicular glutamate transporter2 in ventral posterolateral nucleus of thalamus attenuates neuropathic mechanical allodynia in mice

Gi protein functions in thalamic neurons to decrease orofacial nociceptive response

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