Neonatal hyperoxia alters the host response to influenza A virus infection in adult mice through multiple pathways.


Exposing preterm infants or newborn mice to high concentrations of oxygen disrupts lung development and alters the response to respiratory viral infections later in life. Superoxide dismutase (SOD) has been separately shown to mitigate hyperoxia-mediated changes in lung development and attenuate virus-mediated lung inflammation. However, its potential to… (More)
DOI: 10.1152/ajplung.00112.2013


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