NSAID Use and Risk of Hepatocellular Carcinoma and Intrahepatic Cholangiocarcinoma: The Liver Cancer Pooling Project.

@article{Petrick2015NSAIDUA,
  title={NSAID Use and Risk of Hepatocellular Carcinoma and Intrahepatic Cholangiocarcinoma: The Liver Cancer Pooling Project.},
  author={Jessica L Petrick and Vikrant V. Sahasrabuddhe and Andrew T. Chan and Michael C. R. Alavanja and Laura E. Beane-Freeman and Julie E. Buring and Jie Chen and Dawn Q Chong and Neal D Freedman and Charles S. Fuchs and J. Michael Gaziano and Edward L. Giovannucci and Barry I. Graubard and Albert R. Hollenbeck and Lifang Hou and Eric J Jacobs and Lindsay Y. King and Jill Koshiol and I K Lee and Martha S. Linet and J R Palmer and Mark P Purdue and Lynn A Rosenberg and Catherine Schairer and Howard D Sesso and Alice J. Sigurdson and Jean Wactawski-Wende and Anne Zeleniuch-Jacquotte and Peter T. Campbell and Katherine A McGlynn},
  journal={Cancer prevention research},
  year={2015},
  volume={8 12},
  pages={1156-62}
}
Chronic inflammation plays a pivotal role in the pathogenesis of hepatocellular carcinoma (HCC) and intrahepatic cholangiocarcinoma (ICC), the two most common types of liver cancer. A number of prior experimental studies have suggested that nonsteroidal anti-inflammatory drugs (NSAIDs), including aspirin and ibuprofen, may potentially protect against liver cancer. However, no observational study has examined the association between aspirin duration and dose or other over-the-counter non-aspirin… CONTINUE READING
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