NO and NO-independent mechanisms mediate ETB receptor buffering of ET-1-induced renal vasoconstriction in the rat.

@article{Just2005NOAN,
  title={NO and NO-independent mechanisms mediate ETB receptor buffering of ET-1-induced renal vasoconstriction in the rat.},
  author={Armin Just and Andrea J. M. Olson and John R Falck and William J. Arendshorst},
  journal={American journal of physiology. Regulatory, integrative and comparative physiology},
  year={2005},
  volume={288 5},
  pages={
          R1168-77
        }
}
  • Armin Just, Andrea J. M. Olson, +1 author William J. Arendshorst
  • Published in
    American journal of…
    2005
  • Chemistry, Medicine
  • Vascular endothelin (ET) type B (ET(B)) receptors exert dilator and constrictor actions in a complex interaction with ET(A) receptors. We aimed to clarify the presence and relative importance of nitric oxide (NO) and other mechanisms underlying the dilator effects of ET(B) receptors in rat kidneys. Complete inhibition of NO production with Nomega-nitro-L-arginine methyl ester (L-NAME, 25 mg/kg iv) enhanced the renal vasoconstriction elicited by ET-1 injected into the renal artery from -15 to… CONTINUE READING

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