NNZ-2566 treatment inhibits neuroinflammation and pro-inflammatory cytokine expression induced by experimental penetrating ballistic-like brain injury in rats

@article{Wei2009NNZ2566TI,
  title={NNZ-2566 treatment inhibits neuroinflammation and pro-inflammatory cytokine expression induced by experimental penetrating ballistic-like brain injury in rats},
  author={Hans H Wei and Xi-chun May Lu and Deborah A. Shear and Anu Waghray and Changping Yao and Frank C. Tortella and Jitendra R. Dave},
  journal={Journal of Neuroinflammation},
  year={2009},
  volume={6},
  pages={19 - 19}
}
Inflammatory cytokines play a crucial role in the pathophysiology of traumatic brain injury (TBI), exerting either deleterious effects on the progression of tissue damage or beneficial roles during recovery and repair. NNZ-2566, a synthetic analogue of the neuroprotective tripeptide Glypromate®, has been shown to be neuroprotective in animal models of brain injury. The goal of this study was to determine the effects of NNZ-2566 on inflammatory cytokine expression and neuroinflammation induced… CONTINUE READING
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