Myocardial perfusion, oxidative metabolism, and free fatty acid uptake in patients with hypertrophic cardiomyopathy attributable to the Asp175Asn mutation in the alpha-tropomyosin gene: a positron emission tomography study.

@article{Tuunanen2007MyocardialPO,
  title={Myocardial perfusion, oxidative metabolism, and free fatty acid uptake in patients with hypertrophic cardiomyopathy attributable to the Asp175Asn mutation in the alpha-tropomyosin gene: a positron emission tomography study.},
  author={Helena Tuunanen and Johanna Kuusisto and Jyri O. Toikka and Pertti J{\"a}{\"a}skel{\"a}inen and P{\"a}ivi Marjam{\"a}ki and Keijo J. Peuhkurinen and Tapio Viljanen and Petri Sipola and Kira Q. Stolen and Jarna C Hannukainen and P. Nuutila and Markku Laakso and Juhani Knuuti},
  journal={Journal of nuclear cardiology : official publication of the American Society of Nuclear Cardiology},
  year={2007},
  volume={14 3},
  pages={354-65}
}
BACKGROUND The relationship between myocardial metabolic changes and the severity of left ventricular (LV) hypertrophy in patients with hypertrophic cardiomyopathy (HCM) is largely unknown. We characterized metabolic abnormalities in patients with a genetically identical cause for HCM but with variable LV hypertrophy. METHODS AND RESULTS Eight patients with HCM attributable to the Asp175Asn mutation in the alpha-tropomyosin gene underwent myocardial perfusion, oxidative, and free fatty acid… CONTINUE READING
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