Monosodium glutamate: specific brain lesion questioned.

  title={Monosodium glutamate: specific brain lesion questioned.},
  author={CharlesU. Lowe and Mitchell R. Zavon and John W. Olney and Lawrence G. Sharpe},
  volume={167 3920},

Animal models for analyzing metabolic syndrome‐associated liver diseases

Comparisons of mouse models of obesity and diabetes mellitus and NASH suggest specific and useful characteristics in a suitable model of MS‐associated liver diseases, including hepato‐carcinogenesis, can be highlighted.

‘That Won-Ton Soup Headache’: The Chinese Restaurant Syndrome, MSG and the Making of American Food, 1968–1980

This paperexamines the'discovery'ofthe Chinese restaurant syndrome in 1968 and subsequent reactions by the medical community, scientists, public health authorities and the general public to dangers

Penetration of glutamate into brain of 7-day-old rats

Although three areas with a blood-brain barrier, the cerebral cortex, the hypothalamus and the midbrain, of 7-day-old rats had permeabilities similar to adult rats, the other areas of the brain with a, blood- brain barrier had a permeability about 1.9 times that of adult rats.

Regulation of mouse hepatic CYP2D9 mRNA expression by growth and adrenal hormones.

Findings suggest that an adrenal hormone participates in the regulatory mechanism of CYP2D9 mRNA expression in association with GH.

Dextranase activity in coleoptiles of Avena

Abbey, S. See Maxwell, J. A., et al. Abdel-Gawad, M. See Muir, A. H., Jr., et al. Abell, P. I., et al.: Organic analysis of the returned lunar sample, 757; erratum, 1759 Abelson, P. H. (editorials):

Penetration of Glutamate into Brain of 7-day-old Rats

The greater permeability of the brain of 7-day-old rats may reflect the degree of immaturity of the blood-brain barrier, which is about 1.5-1.9 times that of adult rats.

Human NK cells: their ligands, receptors and functions

The expression, or lack of class I MHC glycoproteins has a marked influence on natural killer cell function and a novel mechanism by which human cytomegalovirus evades immune surveillance by NK cells is documented.