Molecular adaptations in human atrial fibrillation: mechanisms of protein remodelling.

@article{Brundel2001MolecularAI,
  title={Molecular adaptations in human atrial fibrillation: mechanisms of protein remodelling.},
  author={Bianca J. J. M. Brundel and Rob H Henning and Wiek H van Gilst and Isabelle Van Gelder and H. J. G. M. Crijns},
  journal={Netherlands heart journal : monthly journal of the Netherlands Society of Cardiology and the Netherlands Heart Foundation},
  year={2001},
  volume={9 6},
  pages={235-239}
}
The chance that the treatment of atrial fibrillation (AF) is successful depends on the duration of the arrhythmia. The low efficacy of cardioversion therapy after long-term AF can be explained by the occurrence of cellular adaptation mechanisms. In this article we describe ion-channel protein remodelling and structural changes in the atria of patients with persistent and paroxysmal AF and its relation with electrical remodelling. 

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