Mitochondrial enzyme retention by irreversibly damaged rectangular isolated adult rat heart myocytes.

@article{Wenger1985MitochondrialER,
  title={Mitochondrial enzyme retention by irreversibly damaged rectangular isolated adult rat heart myocytes.},
  author={William C. Wenger and Michael P. Murphy and O R Kindig and Charles C. Capen and Gerald P. Brierley and Ruth A. Altschuld},
  journal={Life sciences},
  year={1985},
  volume={37 18},
  pages={
          1697-704
        }
}
4 Citations
Reversible ultrastructural alterations in the myocytic mitochondria of isolated rat hearts induced by oxygen radicals.
TLDR
The cristal adhesions appear to be reversible alterations caused by exposure to oxygen radicals, and were associated with other mitochondrial ultrastructural changes.
Secretion of the Vasoactive Peptides, Endothelin, and Parathyroid Hormone-Related Peptide, by Decidual Explants From Pregnancies Complicated by Intrauterine Growth Restriction
TLDR
The finding that both peptides decreased could not be explained by route of delivery or global tissue dysfunction, and investigation of potential mediators of decidual ET and PTHrP will be necessary to determine why these hormones are both decreased in pregnancies complicated by IUGR.

References

SHOWING 1-10 OF 16 REFERENCES
Contracture in Isolated Adult Rat Heart Cells: Role of Ca2+, ATP, and Compartmentation
TLDR
Three new insights into the contracture process were gained: in the quiescent cell system, the rate of onset of contracture was independent of external Cn2+, supporting the view that the Ca2+ dependence of the rates of onset in the whole heart is related to beat-dependent substrate utilization.
Release of Enzymes from Adult Rat Heart Myocytes
TLDR
The results suggest that simultaneous and complete release of soluble cytoplasmic enzymes occurs as each individual cell sustains sarcolemmal damage.
Ischemic tissue injury.
TLDR
The subendocardial to subepicardial gradient in the severity of ischemia following acute coronary occlusion is described and the failure of cell volume regulation in cells which have entered an irreversible state of ischemic injury is demonstrated by the use of free-hand slices in vitro.
Canine Myocardial Reperfusion Injury: Its Reduction by the Combined Administration of Superoxide Dismutase and Catalase
TLDR
The results of this investigation demonstrate that the ‘primary’ myqcardial cellular damage due to ischemia is additive to the cardiac cell damage during the phase of reperfusion, and that the“secondary” effects are mediated by toxic metabolites of oxygen.
ENZYMATIC PROPERTIES OF THE INNER AND OUTER MEMBRANES OF RAT LIVER MITOCHONDRIA
TLDR
The inner membrane-matrix fraction retained a high degree of morphological and biochemical integrity and exhibited a high respiratory rate and respiratory control when assayed in a sucrose-mannitol medium containing EDTA.
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