MicroRNA-200c contributes to injury from transient focal cerebral ischemia by targeting Reelin.


BACKGROUND AND PURPOSE MicroRNA (miR)-200c increases rapidly in the brain after transient cerebral ischemia but its role in poststroke brain injury is unclear. Reelin, a regulator of neuronal migration and synaptogenesis, is a predicted target of miR-200c. We hypothesized that miR-200c contributes to injury from transient cerebral ischemia by targeting… (More)
DOI: 10.1161/STROKEAHA.114.007041


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