Metformin inhibits HaCaT cell viability via the miR-21/PTEN/Akt signaling pathway.


Substantial preclinical evidence has indicated out a direct anti‑proliferation effect of metformin on various solid tumors; however, further and more detailed exploration into its molecular mechanism remains to be performed. The present study aimed to investigate the effect of metformin on cell viability and its underlying mechanism, in the cultured human… (More)
DOI: 10.3892/mmr.2017.8364


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