Memory following cholinergic (NBM) and noradrenergic (DNAB) lesions made singly or in combination: potentiation of disruption by scopolamine.

Abstract

Groups of rats were trained on either delayed matching or nonmatching to position tasks, then divided into four subgroups and given the following bilateral lesions: (a) SHAM [vehicle injection into the nucleus basalis magnocellularis (NBM) and dorsal noradrenergic bundle (DNAB)], (b) DNAB (6-hydroxydopamine lesion of the DNAB, vehicle into the NBM), (c) NBM… (More)

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@article{Sahgal1990MemoryFC, title={Memory following cholinergic (NBM) and noradrenergic (DNAB) lesions made singly or in combination: potentiation of disruption by scopolamine.}, author={Arjun Sahgal and Ashley B Keith and Selvyn Lloyd and J M Kerwin and Elaine K. Perry and James A. Edwardson}, journal={Pharmacology, biochemistry, and behavior}, year={1990}, volume={37 4}, pages={597-605} }