Membrane physical state controls the signaling mechanism of the heat shock response in Synechocystis PCC 6803: identification of hsp17 as a "fluidity gene".

@article{Horvth1998MembranePS,
  title={Membrane physical state controls the signaling mechanism of the heat shock response in Synechocystis PCC 6803: identification of hsp17 as a "fluidity gene".},
  author={Istv{\'a}n Horv{\'a}th and Attila Glatz and Vikt{\'o}ria Varvasovszki and Zsolt T{\"o}r{\"o}k and Tibor P{\'a}li and G{\'a}bor Balogh and Erika Kov{\'a}cs and L{\'a}szl{\'o} N{\'a}dasdi and S M Benk{\"o} and Ferenc Jo{\'o} and Laszlo Vigh},
  journal={Proceedings of the National Academy of Sciences of the United States of America},
  year={1998},
  volume={95 7},
  pages={3513-8}
}
The fluidity of Synechocystis membranes was adjusted in vivo by temperature acclimation, addition of fluidizer agent benzyl alcohol, or catalytic lipid hydrogenation specific to plasma membranes. The reduced membrane physical order in thylakoids obtained by either downshifting growth temperature or administration of benzyl alcohol was paralleled with enhanced thermosensitivity of the photosynthetic membrane. Simultaneously, the stress-sensing system leading to the cellular heat shock (HS… CONTINUE READING