Mediation of immune glomerular injury.

@article{Couser1990MediationOI,
  title={Mediation of immune glomerular injury.},
  author={William G. Couser},
  journal={Journal of the American Society of Nephrology : JASN},
  year={1990},
  volume={1 1},
  pages={13-29}
}
This paper reviews current concepts of glomerular immune injury of both inflammatory and noninflammatory types. In noninflammatory lesions induced by antibody alone or C5b-9, the glomerular epithelial cell appears to be the principal target of injury. Similar mechanisms are probably operative in human diseases such as minimal change nephrotic syndrome and membranous nephropathy. In inflammatory lesions, circulating effector cells including neutrophils, macrophages, platelets, and probably… CONTINUE READING
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