Corpus ID: 55675435

Mecanismos de acción de los efectos protectores de los estrógenos sobre el sistema cardiovascular

@article{Fernndez2009MecanismosDA,
  title={Mecanismos de acci{\'o}n de los efectos protectores de los estr{\'o}genos sobre el sistema cardiovascular},
  author={V. Fern{\'a}ndez and Cristina Lemini and Yanira Franco},
  journal={Revista de la Facultad de Medicina},
  year={2009},
  volume={46},
  pages={101-108}
}
NUMEROSOS ESTUDIOS CLINICOS SENALAN QUE LOS HOMBRES SON MAS SUSCEPTIBLES A SUFRIR ENFERMEDADES CARDIOVASCULARES QUE LAS MUJERES EN LA MISMA EDAD. LA PROTECCION EN LAS MUJERES ESTA ESTRECHAMENTE RELACIONADA CON LOS NIVELES DE LOS ESTROGENOS EN SANGRE, YA QUE CUANDO LA PRODUCCION DE ESTAS HORMONAS CESA, AUMENTA EL RIESGO DE PADECER ESTE TIPO DE ENFERMEDADES DE MANERA SEMEJANTE AL DE LOS HOMBRES. SE HA OBSERVADO QUE LA TERAPIA DE REEMPLAZO HORMONAL EN MUJERES POSMENOPAUSICAS DISMINUYE ESTE RIESGO… Expand
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References

SHOWING 1-10 OF 62 REFERENCES
[The versatility of estrogens is taken advantage of in the treatment of postmenopause].
La terapia hormonal de reemplazo en la mujer posmenopausica ha demostrado de manera convincente su beneficio para prevenir trastornos cardiovasculares, osteoporosis y probablemente enfermedad deExpand
Cardiovascular effects of estrogen and lipid-lowering therapies in postmenopausal women.
TLDR
Encouraging reports from population-based observational studies of favorable effects of estrogen therapy on cardiovascular morbidity and mortality have led to enthusiasm for widespread use of estrogen by postmenopausal women for prevention of cardiovascular disease events. Expand
Estrogen replacement therapy and coronary heart disease: A quantitative assessment of the epidemiologic evidence☆☆☆
TLDR
The bulk of the evidence strongly supports a protective effect of estrogens that is unlikely to be explained by confounding factors and is consistent with the effect of Estrogen on lipoprotein subfractions. Expand
Cardiovascular mortality and noncontraceptive use of estrogen in women: results from the Lipid Research Clinics Program Follow-up Study.
TLDR
A cohort of 2270 white women, aged 40-69 years at baseline, were followed for an average of 8.5 years in the Lipid Research Clinics Program Follow-up Study and the prevalence of cardiovascular disease at baseline was slightly higher in estrogen users than in nonusers; furthermore, the exclusion of all women with prevalent cardiovascular Disease at baseline did not alter the apparent protective effect of estrogen. Expand
Thrombin enhances the release of endothelin from cultured porcine aortic endothelial cells.
TLDR
Le cycloheximide, inhibiteur de la synthese proteique, inhibe la liberation d'endotheline aussi bien dans les conditions basales qu'apres stimulation par the thrombine. Expand
Estrogens and atherosclerosis.
TLDR
Beneficial effects of estrogens include direct actions on the vascular endothelium and vascular smooth muscle, leading to a decrease in the expression of adhesion molecules involved in monocyte adhesion to endothelial cells, and to a decreases in certain chemokines involved in Monocyte migration into the subendothelial space. Expand
Influence of Sex Hormones on Plasma Endothelin Levels
TLDR
It was found that plasma endothelin levels tended to be higher in men than in women and lower in pregnant women than in nonpregnant controls and in patients with myocardial infarction and diabetes. Expand
Estrogen-induced uterine vasodilatation is antagonized by L-nitroarginine methyl ester, an inhibitor of nitric oxide synthesis.
TLDR
Estradiol-17 beta-induced increases in uterine blood flow are mediated mainly by nitric oxide; the observed vasodilation can be antagonized by the intraaterial administration of nitricoxide synthetase inhibitor L-nitroarginine methyl ester. Expand
Estradiol accelerates functional endothelial recovery after arterial injury.
TLDR
Estrogen accelerates functional endothelial recovery after barotraumatic deendothelializing injury and suggests that the antiatherogenic action of estrogen may be mediated in part through direct effects on endothelial cells. Expand
Sex hormones and coronary disease: a review of the clinical studies
TLDR
Research findings have disputed the hypothesis that persons with coronary disease have low levels of a protective factor such as estrogen or progesterone and high levels of testosterone, and suggested that natural estrogens are antiatherogenic. Expand
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