Maternal protein restriction leads to enhanced hepatic gluconeogenic gene expression in adult male rat offspring due to impaired expression of the liver X receptor.

@article{Vo2013MaternalPR,
  title={Maternal protein restriction leads to enhanced hepatic gluconeogenic gene expression in adult male rat offspring due to impaired expression of the liver X receptor.},
  author={Thin Xuan Vo and Andrew Revesz and Gurjeev Sohi and Noelle L Ma and Daniel B Hardy},
  journal={The Journal of endocrinology},
  year={2013},
  volume={218 1},
  pages={85-97}
}
Epidemiological studies demonstrate that the link between impaired fetal development and glucose intolerance in later life is exacerbated by postnatal catch-up growth. Maternal protein restriction (MPR) during pregnancy and lactation in the rat has been previously demonstrated to lead to impaired glucose tolerance in adulthood, however the effects of protein restoration during weaning on glucose homeostasis are largely unknown. Recent in vitro studies have identified that the liver X receptor… CONTINUE READING
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