Maternal, placental, and fetal pathophysiology of cocaine exposure during pregnancy.

@article{Plessinger1993MaternalPA,
  title={Maternal, placental, and fetal pathophysiology of cocaine exposure during pregnancy.},
  author={Mark A Plessinger and James R Woods},
  journal={Clinical obstetrics and gynecology},
  year={1993},
  volume={36 2},
  pages={267-78}
}
As should be evident from the case reports, epidemiology studies, and animal data, the primary mode of cocaine action is vasoconstriction, whether it occurs on the maternal side or the fetal side of the placenta. Attempts to characterize the fetal effects of cocaine exposure during pregnancy in order to formulate a "fetal cocaine syndrome" has revealed a wide spectrum of fetal effects. Therefore, a well-defined "fetal cocaine syndrome" does not exist. In fact, most fetal defects may be related… CONTINUE READING

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While repeated exposures during pregnancy may increase the risk of cocaine - induced damage , it appears that even a single exposure may produce infarction , edema , and tissue necrosis .
While repeated exposures during pregnancy may increase the risk of cocaine - induced damage , it appears that even a single exposure may produce infarction , edema , and tissue necrosis .
While repeated exposures during pregnancy may increase the risk of cocaine - induced damage , it appears that even a single exposure may produce infarction , edema , and tissue necrosis .
While repeated exposures during pregnancy may increase the risk of cocaine - induced damage , it appears that even a single exposure may produce infarction , edema , and tissue necrosis .
While repeated exposures during pregnancy may increase the risk of cocaine - induced damage , it appears that even a single exposure may produce infarction , edema , and tissue necrosis .
While repeated exposures during pregnancy may increase the risk of cocaine - induced damage , it appears that even a single exposure may produce infarction , edema , and tissue necrosis .
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