Lung inflammation induced by lipoteichoic acid or lipopolysaccharide in humans.

@article{Hoogerwerf2008LungII,
  title={Lung inflammation induced by lipoteichoic acid or lipopolysaccharide in humans.},
  author={Jacobien J Hoogerwerf and Alex F de Vos and P. Bresser and Jaring S van der Zee and Jennie M Pater and A. D. de Boer and M. Tanck and Daniel L Lundell and Chung Her-Jenh and C. Draing and S. von Aulock and T. van der Poll},
  journal={American journal of respiratory and critical care medicine},
  year={2008},
  volume={178 1},
  pages={
          34-41
        }
}
  • Jacobien J Hoogerwerf, Alex F de Vos, +9 authors T. van der Poll
  • Published 2008
  • Medicine
  • American journal of respiratory and critical care medicine
RATIONALE Recognition of pathogen-associated molecular patterns by Toll-like receptors (TLRs) is considered to be important for an appropriate immune response against pathogens that enter the lower airways. OBJECTIVES We studied the effects of two different TLR agonists relevant for respiratory infections in the human lung: lipoteichoic acid (LTA; TLR2 agonist, component of gram-positive bacteria) and lipopolysaccharide (LPS; TLR4-agonist, component of gram-negative bacteria). METHODS… Expand
Priming of alveolar macrophages upon instillation of lipopolysaccharide in the human lung.
Endogenous MCP-1 promotes lung inflammation induced by LPS and LTA.
Divergent roles of Toll‐like receptor 2 in response to lipoteichoic acid and Staphylococcus aureus in vivo
  • Mark R. Gillrie, L. Zbytnuik, +7 authors M. Ho
  • Biology, Medicine
  • European journal of immunology
  • 2010
Differences in lipopolysaccharide- and lipoteichoic acid-induced cytokine/chemokine expression
Activation of coagulation and inhibition of fibrinolysis in the human lung on bronchial instillation of lipoteichoic acid and lipopolysaccharide*
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