Loss of TLR3 and its downstream signaling accelerates acinar cell damage in the acute phase of pancreatitis.

@article{Regel2019LossOT,
  title={Loss of TLR3 and its downstream signaling accelerates acinar cell damage in the acute phase of pancreatitis.},
  author={Ivonne Regel and Susanne Raulefs and Simone Benitz and Charlotte Mihaljevic and Simon Rieder and Georg Leinenkugel and Katja Steiger and Anna Melissa Schlitter and Irene Esposito and Julia Mayerle and Bo Kong and J{\"o}rg Kleeff and Christoph W Michalski},
  journal={Pancreatology : official journal of the International Association of Pancreatology (IAP) ... [et al.]},
  year={2019},
  volume={19 1},
  pages={
          149-157
        }
}
  • I. Regel, S. Raulefs, C. Michalski
  • Published 1 June 2016
  • Biology, Medicine
  • Pancreatology : official journal of the International Association of Pancreatology (IAP) ... [et al.]
Toll‐like receptor 3 expression in myeloid cells is essential for efficient regeneration after acute pancreatitis in mice
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Myeloid Differentiation Primary Response 88–Cyclin D1 Signaling in Breast Cancer Cells Regulates Toll-Like Receptor 3-Mediated Cell Proliferation
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This study provides first comprehensive evidence on the involvement of canonical signaling of TLR3 using MyD88–cyclin D1-mediated breast cancer cell proliferation and will provide valuable insights into understanding theTLR3-mediated adjuvant therapy in cancer.

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