Liposome fluidity alters interactions between the ganglioside GM1 and cholera toxin B subunit.

Abstract

Cholera toxin is a complex protein with a biologically active protein (A subunit) and a cell targeting portion (B subunit). The B subunit is responsible for specific cell binding and entry of the A subunit. One way to limit potential toxicity of the toxin after exposure is to introduce cellular decoys to bind the toxin before it can enter cells. In this… (More)
DOI: 10.1080/08982100801893929

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