Lipoprotein lipase deficiency with pancreatitis in mink: biochemical characterization and pathology.

@article{Christophersen1997LipoproteinLD,
  title={Lipoprotein lipase deficiency with pancreatitis in mink: biochemical characterization and pathology.},
  author={Bj\orn O. Christophersen and Knut Nordstoga and Yuan Shen and Thomas Olivecrona and Gunilla Olivecrona},
  journal={Journal of lipid research},
  year={1997},
  volume={38 5},
  pages={837-46}
}
A severe hyperlipemia in mink, with a pattern that suggested recessive inheritance, was observed at a farm in Norway. On a normal mink diet, affected animals had grossly elevated levels of plasma triglycerides which decreased towards normal on a low-fat diet. Normal minks had the main part of their plasma cholesterol in the HDL fraction. Affected minks, although severely hypertriglyceridaemic, had almost normal levels of both LDL and HDL. Affected minks frequently had lipogranulomas in the… CONTINUE READING

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The minks provide a new animal model for studies on pancreatitis induced by hypertriglyceridemia and on lipoprotein metabolism in the lipoprotein lipase - deficient state and show features similar to those found in human hyperlipoproteinemia type I.
The minks provide a new animal model for studies on pancreatitis induced by hypertriglyceridemia and on lipoprotein metabolism in the lipoprotein lipase - deficient state and show features similar to those found in human hyperlipoproteinemia type I.
The minks provide a new animal model for studies on pancreatitis induced by hypertriglyceridemia and on lipoprotein metabolism in the lipoprotein lipase - deficient state and show features similar to those found in human hyperlipoproteinemia type I.
The minks provide a new animal model for studies on pancreatitis induced by hypertriglyceridemia and on lipoprotein metabolism in the lipoprotein lipase - deficient state and show features similar to those found in human hyperlipoproteinemia type I.
The minks provide a new animal model for studies on pancreatitis induced by hypertriglyceridemia and on lipoprotein metabolism in the lipoprotein lipase - deficient state and show features similar to those found in human hyperlipoproteinemia type I.
The minks provide a new animal model for studies on pancreatitis induced by hypertriglyceridemia and on lipoprotein metabolism in the lipoprotein lipase - deficient state and show features similar to those found in human hyperlipoproteinemia type I.
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